Wednesday, March 19, 2014

Study shows a protein common in cancers can turn into anti-tumor mechanisms

A study conducted at Stony Brook University stated that a cellular protein known as STAT3 is quite overactive in the majority of human cancers. Now this interference could be turned into antitumor mechanism in cells and hence could control growth of cancer. The report was published in the Proceedings of the National Academy of Sciences. The study was conducted as a progressive step in cancer development and this further have provided with the basis for new methods that help to prevent and cure cancer. The lead author Sumita Bhaduri-McIntosh, MD, PhD also an Assistant Professor in the Departments of Pediatrics and Molecular Genetics and Microbiology at Stony Brook University School of Medicine explains, “Our findings add to the short list of known mechanisms by which a key cellular anti-tumor barrier is breached by STAT3 prior to cancer development. Because STAT3 interferes with this innate anti-tumor mechanism in cells, a necessary step in cancer development.”

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